*Corresponding author:
Camila Paiva Perin, Endodontics Department, Tuiuti University of Paraná - Curitiba - Paraná – BrazilReceived: January 20, 2018; Published: February 01, 2018
DOI: 10.26717/BJSTR.2018.02.000718
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Dental resorptions, conditions in which mineralized dental tissues are eliminated by clastic cells on their surface, persist as a treatment challenge for dentists. Although the causes and mechanism of its occurrence are known, many doubts have been observed in its handling, which motivated the making of this article. Dental resorptions in permanent teeth are pathological and, according to their mechanism of occurrence, can be classified as inflammatory and substitutive [1-3]. Clinically, they are asymptomatic and do not induce pulpal, periapical, and periodontal changes, and is usually the consequence of them [4]. The inflammatory tooth resorption occurs when the cementoblasts are removed from the root surface, resulting in a bare dental surface that allows the installation of clasts units, associated with an inflammatory process induced by the aggressor agent [3,4]. The causes may be multiple, especially pulp necrosis, excessive orthodontic forces and alveolodentary trauma. The factors may or may not be associated [1-3].